Lp(a)

Note: this site is for informational purposes only. To view test results or book a test, use the NHS app in England or contact your GP.

An Lp(a) (lipoprotein(a)) test measures the level of lipoprotein(a), a type of cholesterol-carrying particle, in the blood using a blood sample taken from a vein in the arm. It is used to assess inherited risk of cardiovascular disease, including conditions such as heart disease and stroke.

Also known as 
Lipoprotein little a” 
Formal name 
Lipoprotein (a)

Why get tested?

As part of a targeted screen for cardiovascular disease (coronary artery disease (CAD) and cerebrovascular disease) risk assessment.

When to get tested?

Your doctor may request Lp(a) measurement if you have a family history of premature cardiovascular disease or elevated Lp(a) or if you develop cardiovascular disease at a young age, particularly in the absence of conventional risk factors

Sample required?

A blood sample taken from a vein in your arm

Test preparation needed?

No test preparation is needed.

What is being tested?

Lp(a) is a risk factor for heart disease especially when LDL cholesterol is also raised. Lp(a) is a lipoprotein comprising a lipid rich core surrounded by two proteins, apolipoprotein B100 and apolipoprotein (a).

Lp(a) may accelerate atherosclerotic damage (atheroma) by increasing the size of plaque/​atheroma in artery walls. It is retained in the artery wall more than LDL cholesterol as it binds to the artery lining through apolipoprotein (a). Lp(a) is also thought to increase risk of heart attacks by interfering with clotting mechanisms and therefore promoting clot development on the inner surface of blood vessels.

This dual action may explain the role of Lp(a) in the promotion of cardiovascular disease (CVD).

Lp(a) concentrations within the blood are genetically determined and will remain fairly constant in an individual over a lifetime. Concentration is not affected by diet, exercise, and other lifestyle modifications used to lower lipids within the blood. Lp(a) concentrations are slightly lower in men than in women and increase slightly in women after the menopause. The concentration of Lp(a) also varies with ethnicity: patients of African American descent can have concentrations up to 4 times higher than Caucasians, but they may not have a higher risk for CAD.

Common questions